Thrombosis in COVID‐19 patients
This special issue belongs to |
Research & Reviews: Journal of Oncology and Hematology |
Related section |
NA |
Deadline for Manuscript Submission |
March 31st, 2023 |
Deadline for Publication |
April 15, 2023 |
Special Issue Description
Dear Collegues,
Covid sickness 2019 (COVID-19) habitually brings about a hypercoagulable expression that is unequivocally connected with mortality. Microvascular and macrovascular thromboembolic or in situ thrombotic complexities have been seen in COVID-19 in the vasculature of the lungs, spleen, cerebrum, stomach, and periphery. There are reports of regular clots development in hemodialysis circuits, strokes as an introducing highlight in youthful patients who were beforehand sound, and blood vessel and venous thromboembolism arrangement notwithstanding prophylactic or completely helpful anticoagulation.
Besides, thromboses have been recognized both in the intense setting and soon after basic sickness, recommending that the favorable to the thrombotic state could most recent a little while or significantly longer post-hospitalization. Comparable thrombotic entanglements were seen in severe acute respiratory syndrome (SARS) and Middle East Respiratory Syndrome (MERS). Numerous patients with COVID-19 have given intense ischemic stroke or experienced a stroke while hospitalized, including youthful patients under 50 years of age with generally hardly any previous gamble factors. The rate of stroke in ICU patients has been roughly 2.5% in a few COVID-19 cohorts. Mesenteric ischemia, peripheral artery obstruction, and enormous vessel arteriosclerosis obliterans have likewise been reported, as has cerebral venous sinus thrombosis.
Thrombotic complications are successive in COVID-19 and altogether add to mortality and horribleness. As Pulmonary Embolism (PE) has been generally seen in a few examination series, the event of intense PE might make sense of certain instances of unexpected right ventricular dysfunction cardiogenic shock, and abrupt demise notwithstanding troponin rise. Whether long-haul complications, for example, aspiratory hypertension might entangle present COVID recuperation stays to be seen. A few thrombogenic systems are possibly ensnared in COVID-19 apoplexy, including apoplexy set off by cytokine storm, antiphospholipid immune response disorder, macrophage activation syndrome, supplement outpouring, and RAS dysregulation.
Keywords
* COVID-19
* Mortality
* Thrombotic
* Severe acute respiratory syndrome (SARS)
* Middle East Respiratory Syndrome (MERS)
* Cardiogenic Shock
* Pulmonary Embolism (PE)
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