Focus and Scope
About the Journal
International Journal of Cell Biology and Cellular Functions is a peer-reviewed hybrid open-access journal launched in 2023 that aims to stimulate the scientifically grounded exchange of ideas. It contains timely reviews and original research articles, with emphasis on cellular processes and cell biology, including all major fields of research.
Focus and Scope
Cellular microbiology: Generalized transduction, Specialized transduction, Lateral transduction, Process, Bacterial Transduction, Generalized Transduction, Specialized Transduction.
Plant Cell: cell wall, cell membrane, Nucleus, Smooth and rough Endoplasmic Reticulum, Golgi apparatus, ribosomes, plastids, Mitochondria, Vacuoles, vesicles like peroxisomes, Meristematic cells, Parenchyma cells, Collenchyma cells, Sclerenchyma cells, Reproductive cells, Cytoskeleton.
Cell migration: Integrin, Actin, Apoptosis, Cell Proliferation, Endothelial Cells, Extracellular Matrix, Mutation, Proteins, Single-Cell Migration, Collective Cell Migration, Amoeboid movement, Ciliary, Gliding motility, Flagella, Mechanotaxis, Chemotaxis, Cell-matrix adhesions, The Rho family of small GTPases, and Proteases.
Cell Communication: Cell Signaling, Paracrine signaling, Autocrine signaling, Endocrine signaling, Signaling by direct contact, Direct Contact, Reception, Transduction, Response, and Cell Adhesion.
Cell mechanism: Channel Mechanism, Synaptic Mechanism, Ion Concentration, Ligand-gated ion channels. II, Receptors that possess intrinsic guanylyl cyclase activity. III. Receptors with intrinsic tyrosine kinase activity, Cell division, Cellular signaling, Organelle, and membrane structure and function, Gene regulation, localization, Protein structure and function, Proteomics and proteostasis, Multicellular interactions and development.
Gene Regulation: Genes, Gene Expression, Regulation of Gene Expression, Prokaryotic, Eukaryotic Transcription, Regulatory proteins, Chemical modification of DNA, mRNA synthesis (transcription), mRNA degradation, Protein synthesis (translation).
Cellular senescence: Fibroblast, Autophagy, Oxidative Stress, Progenitor Cell, Apoptosis, Phenotype, DNA Damage, Proteins, Reversible senescence, Irreversible senescent, p53, INK4a/Rb, p53 pathways, Primary causes, Antagonistic, Integrative, Features of cell senescence, Oncogene-induced senescence (OIS), Telomere dependent replicative senescence, Programmed senescence, Non-telomeric stress-induced premature senescence, Unresolved DNA damage, Induced senescence, Oxidative stress, Telomere damage/shortening, DNA damage, Mitochondrial dysfunction, Chromatin disruption, Inflammation, Epigenetic dysregulation, Oncogene activation.
Cell-Matrix Interaction: Mesenchymal Stem Cell, Nanotubes, Tissue Engineering, Nanocomposite, Protein, Biomaterial, Integrins, Microenvironments, Transmembrane protein, Material stiffness, Surface chemistry, Surface topography, Adhesion receptors, Multi-protein adhesion, Actin cytoskeleton.
Cell Disruption: Extraction, Homogenization, Centrifugation, Bead mills, Ultrasound, French press, High-pressure homogenizer, Thermolysis, Decompression, Osmotic shock, Detergents, Solvents, Enzymes, Utilization of a Microfluidizer, Liquid Homogenization, Sonication, Freeze-thaw, Manual grinding.
Signal transduction: Adaptor Proteins, Akt Pathway, Calcium-binding Proteins, and Related Molecules, Cellular Senescence and Pathways in Aging, Circadian Rhythm Molecules, G Protein-Coupled Receptors (GPCRs), Intracellular Kinases, ITIM/ITAM Immunoreceptors, and Related Molecules.